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Amphibians & reptiles of Mount Kinabalu (North Borneo) by Rudolf Malkmus, Ulrich Manthey, Gernot Vogel, Peter

By Rudolf Malkmus, Ulrich Manthey, Gernot Vogel, Peter Hoffmann, Joachim Kosuch

Гора Кинабалу, при высоте 4095 м являющаяся наиболее высокой точкой Малайского архипелага, обладает одним из наиболее высоких в мире разнообразием животных и растений. Эта книга представляет современное и полностью иллюстрированное обобщение знаний о герпетофауне данной горы, характеризуя seventy seven видов амфибий и 112 видов рептилий. Она базируется как на результатах обширных полевых исследований, проведенных автором, так и на информации ранее опубликованных работ.
Доп. информация: Файл является рипом с сайта Google Books. Этим определяется отсутствие некоторых страниц (до 20% от общего числа) и невысокое разрешение сканирования (около a hundred dpi).

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67). These results imply that BRCA1 links the detection of DNA damage to the actual repair of the lesion. Depending on the context, BRCA1 may have multiple functions, as it has also been shown to force the damaged cells to engage in cell cycle arrest or apoptosis (60). Another recently identified target of ATM is the Nbs1 protein. Cells from AT and NBS patients have strikingly similar responses to DNA damage, including increased chromosome breakage and radioresistant DNA synthesis, suggesting that ATM and Nbs1 function in the same pathway.

3. Determination of p53 Stability p53 stability measurements can be performed to confirm that the increased p53 protein levels observed in proteasome inhibitor-treated cells results from stabilization of the p53 protein. This section outlines a method that can be used to measure the stability of the p53 protein in untreated and proteasome inhibitor-treated cells. 1. Five separate dishes of cells are exposed to proteasome inhibitors for 5–10 h, as described earlier. Control cells are again exposed to either DMSO or H2O only.

And Kastan, M. B. (1998) The complexity of p53 modulation: emerging patterns from divergent signals. Genes Dev. 12, 2973–2983. 13. Levine, A. J. (1997) P53, the cellular gatekeeper for growth and division. Cell 88, 323–331. 14. El-Deiry, W. , Velculesu, V. , et al. (1993) WAF1, a potential mediator of p53 tumor suppression. Cell 75, 817–825. 15. , and White, E. (1996) The E1B 19K protein blocks apoptosis by interacting with and inhibiting the p53-inducible and death promoting BAX protein. Genes Dev.

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