By Christopher J. Lote (auth.)
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Extra info for Principles of Renal Physiology
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E. if the tubular NaG load is too high, filtration by that nephron is decreased. Further reading Baylis, C. (1986) Glomerular filtration dynamics. In C. J. ), Advances in renal physiology, Croom Helm, London and Sydney, pp. 33-83 Dworkin, L. D. and Brenner, B. M. (1992) Biophysical basis of glomerular filtration In D. W. Seldin and G. Giebisch (eds), The kidney: physiology and pathophysiology, 2nd edition, Raven Press, New York, pp. 979-1016 Haberle, D. A. and Davis, J. M. Tubuloglomerular feedback mechanisms: interrelationships between tubular flow and glomerular filtration rate.
If the tubular NaG load is too high, filtration by that nephron is decreased. Further reading Baylis, C. (1986) Glomerular filtration dynamics. In C. J. ), Advances in renal physiology, Croom Helm, London and Sydney, pp. 33-83 Dworkin, L. D. and Brenner, B. M. (1992) Biophysical basis of glomerular filtration In D. W. Seldin and G. Giebisch (eds), The kidney: physiology and pathophysiology, 2nd edition, Raven Press, New York, pp. 979-1016 Haberle, D. A. and Davis, J. M. Tubuloglomerular feedback mechanisms: interrelationships between tubular flow and glomerular filtration rate.
Consequently, there is a balance between formation and reabsorption of tissue fluid. 5b. The sequence of blood vessels is: afferent arterioles, glomerular capillaries, efferent arterioles, peritubular capillaries, venules and veins. The presence of a second resistance vessel, the efferent arteriole, means that the hydrostatic pressure in the glomerular capillaries falls very little throughout their length. This pressure is slightly higher, at 45 mmHg, than that in the capillaries of most other vascular beds (about 32 mmHg).