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42. 43. 44. 45. 46. 47. 48. 49. 50. 51. 52. 53. 54. 55. 21 immunohistochemical methods. Am J Respir Cell Mol Biol 1995;12:589–96. Forkert PG. 1,1-Dichloroethylene-induced Clara cell damage is associated with in-situ formation of the reactive epoxide: immunohistochemical detection of its glutathione conjugate. Am J Respir Cell Mol Biol 1999;20:1310–18. Forkert PG, Millen B, Lash LH, et al. Pulmonary bronchiolar cytotoxicity and formation of dichloroacetyl lysine protein adducts in mice treated with trichloroethylene.
Cell-specific expression of CYP2A5 in the mouse respiratory tract: effects of olfactory toxicants. J Histochem Cytochem 2003;51:1545–55. Putt DA, Ding X, Coon MJ, Hollenberg PF. Metabolism of aflatoxin B1 by rabbit and rat nasal mucosa microsomes and purified cytochrome P450, including isoforms 2A10 and 2A11. Carcinogenesis 1995;16:1411–17. Ding X, Coon MJ. Immunochemical characterization of multiple forms of cytochrome P-450 in rabbit nasal microsomes and evidence for tissue-specific expression of P-450s NMa and NMb.
Note the focal area of consolidation in the right lower lobe medially. drug therapy. 23 With greater degrees of haemorrhaging, areas of consolidation may also be seen (Fig. 8). 23 NON-CARDIOGENIC PULMONARY OEDEMA NCPE is a less common pattern of drug-induced lung involvement. This typically occurs acutely with a close temporal relationship between exposure to the drug and onset of dyspnoea and radiographic changes. 24 On occasion, there can be histologic overlap with some of the features of DAD.